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Because it escapes binding to these inhibitory proteins, IGF-1 LR3 remains free in experimental environments to interact continuously with the IGF-1 receptor, resulting in an extended biological half-life and vastly increased potency compared to standard IGF-1

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For long‐term consideration, recent pilot studies found that chronic VNS therapy led to a significant reduction in several plasma cytokines after 6 months to 4 years of treatment (Lespérance et al